There is absolutely no gender marker for transsexualism.
In fact, a study published just 3 weeks ago further supports the research given on my website, sexchangeregret.com and the research referenced in my books: no biological basis can be found for transsexualism.
I see a troubling uptick in misinformation—actual lies— being told in the media to try to convince us that little kids with gender confusion need to take hormone blockers. But there is no such thing as being trapped in the wrong body.
We need to stop "transgender affirming" and tell those who suffer from gender issues the truth.
Transsexualism is a home-grown, depressive mental disorder.
Here is the abstract of the study, taken from http://www.ncbi.nlm.nih.gov/pubmed/25124466.
J Sex Med. 2014 Aug 15. doi: 10.1111/jsm.12673. [Epub ahead of print]
Association Study of ERβ, AR, and CYP19A1 Genes and MtF Transsexualism.
Fernández R1, Esteva I, Gómez-Gil E, Rumbo T, Almaraz MC, Roda E, Haro-Mora JJ, Guillamón A, Pásaro E.1Departamento de Psicobiología, Universidad de A Coruña, A Coruña, Spain.
The etiology of male-to-female (MtF) transsexualism is unknown. Both genetic and neurological factors may play an important role.
To investigate the possible influence of the genetic factor on the etiology of MtF transsexualism.
We carried out a cytogenetic and molecular analysis in 442 MtFs and 473 healthy, age- and geographical origin-matched XY control males. The karyotype was investigated by G-banding and by high-density array in the transsexual group. The molecular analysis involved three tandem variable regions of genes estrogen receptor β (ERβ) (CA tandem repeats in intron 5), androgen receptor (AR) (CAG tandem repeats in exon 1), and CYP19A1 (TTTA tandem repeats in intron 4). The allele and genotype frequencies, after division into short and long alleles, were obtained.
MAIN OUTCOME MEASURES:
We investigated the association between genotype and transsexualism by performing a molecular analysis of three variable regions of genes ERβ, AR, and CYP19A1 in 915 individuals (442 MtFs and 473 control males).
Most MtFs showed an unremarkable 46,XY karyotype (97.96%). No specific chromosome aberration was associated with MtF transsexualism, and prevalence of aneuploidy (2.04%) was slightly higher than in the general population. Molecular analyses showed no significant difference in allelic or genotypic distribution of the genes examined between MtFs and controls. Moreover, molecular findings presented no evidence of an association between the sex hormone-related genes (ERβ, AR, and CYP19A1) and MtF transsexualism.